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Are BTK and PLCG2 mutations essential and sufficient for ibrutinib resistance in chronic lymphocytic leukemia?
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mutations provided The reality that, as stated under, CLL therapy is predicated on the existence or absence of those mutations. The existing consensus MBL77 is always that, apart from clonal mutations, subclonal mutations using a variant allelic frequency ranging from five to ten% (and therefore beneath the brink of detection by typical molecular approaches) could also be noted, While These which has a variant allelic frequency reduce than 5% must not, but there's Substantially controversy all around these challenges and this suggestion may possibly modify Sooner or later.
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